Abstract

1. Abstract
1.1. Case
A 32 year-old male presented to clinic with low-energy bilateral patellar tendon ruptures sustained 3 days apart. Further workup revealed significantly elevated parathyroid hormone (PTH) and calcium levels consistent with parathyroid adenoma. In conjunction with endocrine surgery, an en-bloc resection of his adenoma was performed followed by bilateral tendon repair. His postoperative course was complicated by rebound hypocalcemia, colloquially known as “hungry bone syndrome”.